题目
题目
单项选择题

It can be inferred from the results of the telomerase reactivation in aged telomerase-deficient mice (TERT-ER mouse) that telomerase reactivation in humans could: Not significantly reverse tissue degeneration, as it is not a main factor in aging Reduce tissue degeneration with surprisingly few side effects, as they behaved like stem cells Have rejuvenating effects, but increase the risk of cancer Causes more rapid tissue degeneration, as a longer telomerase is associated with aged cells. A 59-year-old man is found to have elevated IGF-1 levels, which helped him build muscle mass and maintain fertility earlier in life. However, his doctor now warns him about higher risks of cancer and metabolic disease. Which combination of aging theories best explains this tradeoff?

选项
A.Telomere Shortening Theory + Wear and Tear Theory
B.Free Radical Theory + Disposable Soma Theory
C.Antagonistic Pleiotropy Theory + Endocrine/Clock Theory
D.Error Catastrophe Theory + Cross-Linakage Theory
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思路分析
First, restating the question helps frame what we’re evaluating: It asks about what the telomerase reactivation results in aged, telomerase-deficient mice imply for humans, and then a separate scenario about aging theories explaining a tradeoff between IGF-1–related muscle-building and cancer/metabolic disease risk. Option 1: Telomere Shortening Theory + Wear and Tear Theory. The telomere shortening theory focuses on telomere length as a driver of aging, while the wear-and-tear theory emphasizes cumulative damage over time. While......Login to view full explanation

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